4 edition of Life-threatening arrhythmias during ischemia and infarction found in the catalog.
Includes bibliographies and index.
|Statement||editors, David J. Hearse, Allan S. Manning, Michiel J. Janse.|
|Contributions||Hearse, David J., Manning, Allan S., Janse, Michiel Johannes.|
|LC Classifications||RC685.C173 L54 1987|
|The Physical Object|
|Pagination||x, 229 p. :|
|Number of Pages||229|
|LC Control Number||86042585|
Arrhythmias can also produce a heart rate that is too slow (bradycardia—under 60 beats a minute) or too fast (tachycardia—over beats a minute at rest). Most common arrhythmias. Life-Threatening Ventricular Arrhythmias Due to Transient or Correctable brillators in patients with life-threatening sustained ventric-See page ular arrhythmias (1–3). Patients qualiﬁed for randomization in new Q-wave myocardial infarction (MI), new non–Q-wave MI, other ischemic event, proarrhythmic Cited by: changes of ischemia or for QT interval prolongation (if no proarrhythmic drugs are being administered); however, a patient with acute myocardial infarction (MI) given an antiarrhythmic agent requires monitoring for arrhythmias, ischemia, and QT prolongation. Although the need for skilled healthcare professionals with.
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Life-Threatening Arrhythmias During Ischemia and Infarction: Medicine & Health Science Books @ ed by: `This book will provide the cardiologist valuable information on the various mechanisms of lethal arrhythmias arising from myocardial ischemia and infarction.' Acta Price: $ Life-threatening arrhythmias during ischemia and infarction.
New York: Raven Press, © (OCoLC) Online version: Life-threatening arrhythmias during ischemia and infarction. New York: Raven Press, © (OCoLC) Document Type: Book: All Authors / Contributors: David J Hearse; Allan S Manning; Michiel Johannes Janse.
Lethal Arrhythmias Resulting from Myocardial Ischemia and Infarction Proceedings of the Second Rappaport Symposium. Editors: Rosen, Michael R., Palti, Yoram (Eds.) Free Preview. Silent ischemic events were associated with the initiation of life-threatening ventricular arrhythmias in five patients with induced or spontaneous focal coronary artery spasm (or both).Cited by: How the head rules the heart, in Life Threatening Arrhythmias During Ischemia and Infarction, D.J.
Hearse, A.S. Manning, and M Arrhythmias in the Early Ischemic Period. In: Rosen M.R., Palti Y. (eds) Lethal Arrhythmias Resulting from Myocardial Ischemia and Infarction. Developments in Cardiovascular Medicine, vol Springer, Boston, by: 1. These results suggest that a subclinical mutation in SCN5A resulting in a loss of function may predispose to life-threatening arrhythmias during acute ischemia.
In another cohort of patients who developed long QT intervals and Torsade de Pointes (TdP) arrhythmias in days 2–11 following an AMI, a genetic screen of all long QT genes was by: Journals & Books; Register Sign in.
The intention of this review has been to summarise the current state of knowledge regarding the arrhythmias induced by myocardial ischaemia and infarction. Both clinical and experimental aspects were considered. There has been some progress toward understanding the electrophysiological mechanisms Cited by: Ischemia and Myocardial Infarction.
Type A acute aortic dissection is a life-threatening vascular emergency because of its high morbidity and mortality. hypoxemia, myocardial ischemia. Significant ST-segment depression can occur during AVNRT, and represents repolarization changes rather than myocardial ischemia.
Treating the rhythm needs to be divided into acute therapy and prevention of recurrence. As with all arrhythmias causing hemodynamic compromise, electrical cardioversion would be appropriate. Supraventricular or ventricular arrhythmias may be present, either as precipitating factor or as a result of ischemia.
It's important to detect signs of diseases that may contribute to or accompany atherosclerotic heart disease, eg DM, xanthelasma, tendinous xanthomas, HTN, thyrotoxicosis, myxedema, or peripheral artery disease.
It can create a life-threatening situation, and the vascular diseases related to ischemia are among the first causes of mortality and morbidity in the world.
arrhythmias resulting from. Acute-myocardial infarction (AMI)-induced ventricular fibrillation (VF) frequently occurs without warning, often leading to death within minutes in patients who do not receive prompt medical attention.
Identification of patients at risk of AMI-induced lethal ventricular arrhythmias remains an unmet medical by: Rationale of Therapy in the Patient with Acute Myocardial Infarction and Life-Threatening Arrhythmias: A Focus on Bretylium BENEDICT R. LUCCHESI, PhD, MD Experimental evidence suggests a number of pathologic and electrophysiologic mechanisms that may help initiate ventricular arrhythmias accom- panying myocardial ischemia and by: 7.
Ventricular arrhythmias during the acute phase of myocardial infarction are common and account for approximately 80 % of sudden cardiac death cases.
A biphasic curve has been observed in various species and possibly applies also in by: 3. The cellular and electrophysiological changes that accompany infarct healing begin early, but can continue for many months, and patients who have suffered a myocardial infarction remain susceptible to ventricular arrhythmia for life.
Early during infarct healing the transmembrane potential, action potential amplitude and duration, and upstroke velocity are moderately reduced, but not to the extent seen during acute ischemia Cited by: 4. The effect of antiarrhythmic drugs on life-threatening arrhythmias induced by the interaction between acute myocardial ischemia and sympathetic hyperactivity Transient myocardial ischemia, with attendant sympathetic hyperactivity, seems to play a major role in sudden cardiac death among patients with ischemic heart by: The effect of antiarrhythmic drugs on life-threatening arrhythmias induced by the interaction between acute myocardial ischemia and sympathetic hyperactivity.
Heart J. – ().Cited by: Life threatening arrhythmias in intracranial hemorrhage Electro- cardiographic changes simulating myocardial ischemia and infarction associated with spontaneous intra- cranial hemorrhage.
Czrculatmn Manning GWElectrocardiographic changes simulating myocardial ischemia and infarction associated with spontaneous Author: Howard S. Schwartz, Philip R. Yarnell. We obtained the recordings promptly, with patients attached to the monitor at a mean time of 7 (median 4) hours from the onset of symptoms, and within 49 (median 44) minutes of arrival to the ED.
Because PVCs commonly develop during ischemia, it would follow that arrhythmias would be more likely to occur early in the patient’s clinical course. Specifically, reperfusion after a brief period of myocardial ischemia can lead to lethal arrhythmias (Tennant and Wiggers ; Wit and Janse ).
In this context, Prinzmetal et al. () first described a variant form of resting, ST segment elevation angina caused by coronary artery by: 5.
The occurrence of life-threatening arrhythmias consisting of VT, VF, asystole, and complete AV block was determined during the hospital stay. VTs lasting >30 s were classified as sustained. Mortality 1 year after initial presentation was obtained by telephone contact with the patient or the treating physician and compared between patients Cited by: challenge) may be required to establish the diagnosis.
Arrhythmias, both ventricular tachyarrhythmias and heart block, can occur during the ischaemic episode. Cardiac syndrome X refers to those patients with a good history of angina, a positive exercise test and angiographically normal coronary arteries. They. Introduction. Ventricular arrhythmias are important causes for sudden cardiac death in acute myocardial infarction.
1 Early initiation of a beta-blocker treatment reduces the occurrence of ventricular fibrillation (VF) in these patients, 2 suggesting that an increased sympathetic activation is likely to play an important role in early mortality during acute myocardial by: Spontaneous MI related to ischemia due to a primary coronary event such as plaque erosion and/or rupture, fissuring, or dissection.
Type 2 MI secondary to ischemia due to either increased oxygen demand or decreased supply, for example, coronary artery spasm, coronary embolism, anemia, arrhythmias, hypertension, or hypotension.
Type 3Author: Apoorva Bhushan, Mayank Kulshreshtha. A comparison of electrocardiographic changes during reperfusion of acute myocardial infarction by thrombolysis or percutaneous transluminal coronary angioplasty.
Am Heart J ; Hackett D, McKenna W, Davies G, Maseri A. Reperfusion arrhythmias are rare during acute myocardial infarction and thrombolysis in man.
Patients are screened for significant arrhythmias and other possibly significant ECG-patterns directly after discharge and two weeks after myocardial infarction using wearable devices.
The home monitoring data will be linked with extensive data from electronic health records collected before, during hospital stay and after discharge. It is known that myocardial ischaemia and infarction leads to severe. metabolic and electrophysiological changes that induce silent or symptomatic life-threatening arrhythmias.
Sudden cardiac death is most often attributed to this pathophysiology, but many patients sur-vive the early stage of an acute coronary syndrome (ACS) reachingCited by: Flecainide increased mortality following myocardial infarction (MI) in the Cardiac Arrhythmias Suppression (CAST) trial (Echt et al., ) in which patients surviving acute MI (and who are characteristically at high risk of further acute MI) were randomized to receive maintenance antiarrhythmic therapy or by: 92 Sinus Bradycardia Physiological variant due to strong vagal tone or atheletic training.
Rate as low as 50 at rest and 40 during sleep. Common causes: Hypothermia, hypothyroidism, Drug therapy with betablockers, digitalis and other antiarrhythmic drugs. Acute ischemia and infarction of the sinus node (as a complication of acute myocardial.
Arrhythmias and arrhythmology. 24 Chapters. Mechanisms of cardiac arrhythmias: from automaticity to re-entry (reentry) Aberrant ventricular conduction (aberrancy, aberration) Myocardial Ischemia & Infarction. 20 Chapters Conduction Defects. 11 Chapters. Cardiac Hypertrophy & Enlargement.
5 Chapters. Nonischemic chest pain, including potentially life-threatening conditions such as aortic dissection, pulmonary embolism, and esophageal rupture (table 1 and table 2A-B) The diagnosis of acute coronary ischemia depends upon the characteristics of the chest pain, specific associated symptoms, abnormalities on electrocardiogram (ECG), and levels.
Geddes LS. What are the effects of potassium on the electrophysiology of acute ischemia. In: Life Threatening Arrhythmias during Ischemia and Infarction Eds. DJ Hearse, AS Manning, MJ Janse. Raven Press, New York, Google ScholarAuthor: David J.
Hearse. This editorial refers to ‘The cardiac sympathetic co-transmitter neuropeptide Y is pro-arrhythmic following ST-elevation myocardial infarction despite beta-blockade’, by M. Kalla et al., doi: /eurheartj/ehz The knowledge that sympathetic activation can, under the correct circumstances, initiate lethal arrhythmias is far from new.
While monomorphic ventricular tachycardia (VT) is not usually due to active ischemia, polymorphic VT or ventricular fibrillation (VF) are often ischemia-induced arrhythmias.
scar from prior myocardial infarction) or the loss of cell-to-cell coupling in patients with dilated cardiomyopathy provides the proper substrate for reentry, the mechanism thought to be.
Life-Threatening Ventricular Arrhythmias in Patients with Silent Myocardial Ischemia Due to Coronary-Artery Spasm This article has no abstract; the first words appear below. Myocardial infarction (MI) refers to tissue death of the heart muscle caused by ischaemia, that is lack of oxygen delivery to myocardial is a type of acute coronary syndrome, which describes a sudden or short-term change in symptoms related to blood flow to the heart.
Unlike the other type of acute coronary syndrome, unstable angina, a myocardial infarction occurs Causes: Usually coronary artery disease. No studies have examined whether initiation of therapies such as statin or aspirin during acute infections mitigates the increased risk of myocardial infarction.
Share via: Clinical Topics: Arrhythmias and Clinical EP, Diabetes and Cardiometabolic Disease, Dyslipidemia, Heart Failure and Cardiomyopathies, Prevention, SCD/Ventricular Arrhythmias.
Request PDF | OnFederico Migliore and others published Myocardial Edema as a Substrate of Electrocardiographic Abnormalities and Life-Threatening Arrhythmias in. Arrhythmia means an irregular heartbeat.
Diagnosing it is critical, as arrhythmia can indicate severe heart damage. Learn about types of arrhythmias, causes, and treatments here.
Cardinal R, Janse MJ, Eeden J van, Werner G, Naumann d’Alnoncourt C, Durrer D: The effects of lidocaine on intracellular and extracellular potentials, activation and ventricular arrhythmias during acute regional ischemia in the intact isolated porcine heart.
Circ Res –, PubMed Google ScholarAuthor: Michiel J. Janse.Ischemia or ischaemia is a restriction in blood supply to tissues, causing a shortage of oxygen that is needed for cellular metabolism (to keep tissue alive).
Ischemia is generally caused by problems with blood vessels, with resultant damage to or dysfunction of also means local anemia in a given part of a body sometimes resulting from constriction (such as .3. Medical therapy. Underlying causes of complex VA should be treated, if possible.
Therapy of congestive heart failure (CHF), digitalis toxicity, hypokalemia, hypomagnesemia, hypertension, LV dysfunction, LV hypertrophy, myocardial ischemia by anti-ischemic drugs such as beta blockers or by coronary revascularization, hypoxia, and other conditions may abolish or decrease .